The purpose of this site is to collect lab research by medical doctors about herbs that are proven to treat illnesses and counter the false attacks on herbs by the medical industry and false claims by alternative medicine. I let the science tell the facts.
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Quercetin pronounced quer-ce-tin

1. alleviates diabetic symptoms, improves liver and pancreas functions, 2, its free radicals and antioxidants improve and protect the heart from streptozotocin and cardiotoxicity.(damage) 3. regulation of immune cells, inhibit a variety of human diseases, including major depression, Alzheimer's disease and cancer. 4. helps KappaB and Sodium Potassium Pump. 5. anti-inflammatory

1. "Dietary quercetin alleviates diabetic symptoms and reduces streptozotocin-induced disturbance of hepatic (liver) gene...Quercetin is a food component that may ameliorate the diabetic symptoms... Dietary quercetin might improve liver and pancreas functions..." ( In other words this report says quercitin may heal and increase the function of the liver and pancreas.)

" a naturally occurring chemical that is particularly toxic to the insulin-producing beta cells of the pancreas..."

2. This report injected male rats with Quercetin and a-tocopherol,(a drug used to treat heart atacks) then caused them to have a heart attack. "quercetin and a-tocopherol exhibited cardioprotective effects against isoproterenol-induced cardiotoxicity due to their scavenging free radicals, improving antioxidants and maintaining Ca(2+) levels" PubMed 21322096

cardioprotective effects "serving to protect the heart <cardioprotective effects of ACE inhibitors> <a cardioprotective agent>"

"Cardiotoxicity is a condition when there is damage to the heart muscle. As a result of cardiotoxicity, your heart may not be able to pump blood through out your body as well."

3. "RESULTS: The rats treated with 20 mg/kg of quercetin had significantly decreased incidence of EAM, Hw/Bw, macroscopic and microscopic scores of hearts. Further, in EAM rats treated with quercetin levels of TNF-a and IL-17 were significantly lower, while the level of IL-10 was significantly higher both in serum and culture supernatants of LNC stimulated with concanavalin A compared with vehicle-treated animals.CONCLUSIONS: The present study suggests that quercetin ameliorates EAM, at least in part, by interfering production of proinflammatory (TNF-a and IL-17) and/or anti-inflammatory (IL-10) cytokines."

EAM stands for "Enterprise asset management" or management of the whole body.

Amelikorates stands for (to make something bad)

TNF "Tumor necrosis factor (TNF, cachexin or cachectin and formerly known as tumor necrosis factor-alpha) is a cytokine involved in systemic inflammation and is a member of a group of cytokines that stimulate the acute phase reaction. The primary role of TNF is in the regulation of immune cells. TNF is able to induce apoptotic cell death, to induce inflammation, and to inhibit tumorigenesis and viral replication. Dysregulation of TNF production has been implicated in a variety of human diseases, including major depression,[1] Alzheimer's disease[2] and cancer.[3] Recombinant TNF is used as an immunostimulant under the INN tasonermin. Tumor necrosis factor-a can be produced ectopically in the setting of malignancy and parallels parathyroid hormone both in causing secondary hypercalcemia and in the cancers with which excessive production is associated."

4."The effect of quercetin, a plant-derived bioflavonoid with documented positive effect on the cardiovascular system,...Quercetin induced a decrease of Na,K-ATPase activity in the presence of all ATP and Na(+) concentrations investigated..." This is an essential bit of research because a very important item in the body is the Sodium Potassium Pump referred to as Na,K-ATPase activity, because it creates an electrical chemical process necessary to regulate the body. Thus Quercetin is important to run these pumps. see our page on the Sodium Pump

"Hyperoside (quercetin-3-O-galactoside) is a flavonoid compound mainly found in the herb plants Hypericum perforatum L and Crataegus pinnatifida...In this study, hyperoside was shown to exert an anti-inflammatory action...In addition, hyperoside inhibited nuclear factor-?B activation and I?B-a degradation. The present study suggests that an important molecular mechanism by hyperoside reduces inflammation, which might explain its beneficial effect in the regulation of inflammatory reactions."

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